RETINAL VEIN OBSTRUCTION

INTRAVITREAL ANTIVEGF/OZURDEX INJECTIONS-MICROPULSE SUBLIMINAL LAZER PHOTOSTIMULATION,PRP GROWTH FACTOR INJECTION AND MAGNOVISION THERAPY

Retinal veins can be blocked in patients with hypertension, diabetes, cardiovascular disease and eye pressure. This occlusion may occur just at the exit from the optic nerve, that is, in the form of retinal root vein occlusion, or it may be in only one branch of the vein.Vascular pressure rises behind the blockage.Bleeding and edema occur in the retina. Blurred and hazy vision occurs, coinciding with the area of ​​bleeding and edema.

Fundus fluorescein angiography, which will be performed after the hemorrhages are partially removed, is used to investigate the presence of ischemia, that is, the nutritional status of the retina.

In the absence of ischemia, outcomes are generally better.

If there is ischemia in retinal vein root occlusion, new blood vessels are formed within 3 months; Depending on these, intraocular bleeding and a painful and difficult-to-treat glaucoma called 100-day glaucoma (neovascular glaucoma) may develop.

Intraocular injection treatments, micropulse or grid/panretinal thermal laser treatments are applied if necessary to prevent new vessel formation and/or macular edema.

If ischemic damage to the retina is extensive, magnovision therapy and extraocular growth factor injection support may be required.

The underlying cause should be investigated; Hypertension, diabetes, glaucoma must be taken under control.Retinal artery (arterial) occlusion.

Occlusion occurs in the artery that feeds the retina, as a result of the formation of fatty plaques or a clot from the carotid artery called the heart or carotid artery.

If the occlusion occurs in the part of the optic nerve, retinal main artery occlusion occurs. Painless and sudden loss of vision occurs.

Its treatment is very urgent. If the clot cannot be dissolved or moved further within 4 hours, permanent vision loss develops.

In the treatment, intraocular pressure is lowered, and subcutaneous low-weight heparin is started.

Vasodilating drugs are given. If possible, start hyperbaric oxygen therapy quickly. Hyperbaric oxygen both provides dilation of retinal vessels and meets the oxygen requirement of the retina. Monovision therapy to be applied as soon as possible and growth factor injection support out of the eye contribute to the repair of retinal damage.

If arterial occlusion occurs in only one branch, retinal ischemia, that is, damage, is seen only in the retinal region supplied by that branch.